# Rebuild Neuropathy Treatment Center — Full Reference 3320 N Federal Hwy, Lighthouse Point, FL 33064 · (754) 547-2350 · https://rebuildneuropathytreatmentcenter.com Neuropathy treatment. In-clinic only. Pricing quoted at consultation. Physician-supervised ## The Rebuild Neuropathy Protocol™ Root-cause diagnosis first, then a modality combination matched to the cause. Not a flat-fee one-size-fits-all program. Most patients complete an initial protocol over 8 to 12 weeks, with maintenance typically quarterly afterward. 1. **Root-cause diagnosis** — The first visit establishes what is damaging the nerve, not merely that something is. We review history, the full medication and supplement list, recent labs, and any prior nerve conduction study or EMG, then examine the affected territories with monofilament, vibration and reflex testing. Where the picture is incomplete, we order what is missing: A1c, B12, a thyroid panel, hormone levels, or imaging when root compression is suspected. Nothing is scheduled until there is a working cause. 2. **Modality matching** — Six modalities are available and most patients need three or four. Compressive findings bring in mechanical decompression with the Antalgic-Trac®. Poor glycemic control and an inflammatory metabolic picture bring in metabolic optimization and IV nerve support. Documented thyroid or sex hormone deficiency brings in hormonal correction. Your clinician explains which modalities were selected and, just as usefully, which were left out and why. 3. **Active treatment** — The initial course typically runs eight to twelve weeks of scheduled in-clinic sessions. Laser and shockwave are applied over the affected nerve territories, infusions are given in clinic, and the metabolic and hormonal components are managed alongside them under physician supervision. Progress is reassessed during the course using the same measures taken at baseline, so change is tracked rather than assumed. If a component is not contributing, it comes out of the plan. 4. **Maintenance** — Peripheral nerves regenerate slowly, and the metabolic conditions that damaged them usually persist. After the initial course, most patients move to a maintenance schedule that is typically quarterly, adjusted to how the nerves have responded. Maintenance is lighter than the initial protocol. Its purpose is to hold the ground gained rather than repeat the full course. **Limits of the protocol.** Conservative regenerative treatment addresses the functional spectrum of neuropathy, which means there has to be surviving nerve tissue to work with. Patients with advanced axonal degeneration from decades of uncontrolled diabetes or long-term chemotherapy may have permanent structural damage that limits the ceiling of functional recovery. End-stage neuropathy with complete loss of sensory or motor function, confirmed on nerve conduction studies showing absent potentials rather than reduced amplitudes, is irreversible structural damage, and this protocol does not reverse complete denervation. We would rather tell you that at consultation than take you through a course of treatment that cannot help. --- # ROOT CAUSES — FULL DETAIL ## Diabetic Neuropathy - URL: https://rebuildneuropathytreatmentcenter.com/conditions/diabetic-neuropathy - Also called: Diabetic peripheral neuropathy - Definition: Diabetic neuropathy is nerve damage caused by prolonged exposure of peripheral nerves to elevated blood glucose. It affects the longest nerves first, which is why symptoms usually start in the toes and move up the feet over months or years. The damage involves both the sensory fibers themselves and the small vessels that feed them, the vasa nervorum. - Common signs: Burning or tingling in the feet that is worse at night; Numbness that makes the floor feel padded or far away; Sharp, electric pains in the toes or soles; Loss of protective sensation on monofilament testing; Unsteadiness in the dark or on uneven ground; Cuts and blisters on the feet that go unnoticed - Causes: Chronic hyperglycemia and years of elevated A1c; Microvascular damage to the vasa nervorum that supply peripheral nerves; Insulin resistance and the inflammatory metabolic state that accompanies it; Coexisting vitamin B12 deficiency, which long-term metformin use can produce - How it is identified: Diabetic neuropathy is confirmed by testing rather than assumed from a diabetes diagnosis. The evaluation includes A1c and fasting glucose, 10-gram monofilament and vibration testing at the feet, and serum B12 with methylmalonic acid, since B12 deficiency both mimics and compounds diabetic nerve damage. Thyroid function is checked because hypothyroid neuropathy is common and correctable. Nerve conduction studies are used when the distribution is asymmetric, when weakness is present, or when the amount of surviving nerve function needs to be quantified before committing to a protocol. - Modalities typically indicated: metabolic-optimization, class-iv-laser-therapy, iv-nerve-support-infusions - Prognosis: How much is recoverable depends on how much axon is left. Patients still in the burning and tingling phase, before protective sensation is lost on monofilament testing, generally have more to work with than those carrying decades of uncontrolled glucose. Where nerve conduction studies show absent potentials rather than reduced amplitudes, the loss is structural and permanent, and no conservative regenerative protocol reverses complete denervation. Your clinician will tell you at consultation which of those descriptions your testing fits. ### Why Diabetic Neuropathy Starts in the Feet Peripheral nerves are metabolically expensive to maintain, and the longest axons, running from the lumbar spine down to the toes, are the most expensive of all. When chronic hyperglycemia damages the vasa nervorum that supply those axons and drives inflammation in the surrounding tissue, the distal ends fail first. That is why the pattern is symmetric and stocking-shaped, appearing in both feet before anything is felt in the hands. A patient who reports burning only in one foot usually has something else going on, which is why we map the distribution carefully. ### Testing Comes Before Any Treatment A diabetes diagnosis explains why neuropathy is likely, not what is currently driving it. Two patients with the same A1c can have very different findings on monofilament and vibration testing, and one of them may also be running a B12 deficiency that is doing its own damage. We establish the mechanism and the remaining nerve function first, then build the protocol around it. That is the difference between the Rebuild Neuropathy Protocol™ and a flat-fee package sold before anyone has examined your feet. ### How the Protocol Targets Diabetic Nerve Damage Metabolic optimization addresses the environment the nerve lives in, using physician-prescribed semaglutide along with dietary guidance to reduce the inflammatory metabolic state that drives progressive damage. Peripheral neuropathy is not an FDA-approved indication for semaglutide; it is prescribed here for the metabolic driver rather than for the nerve itself. LightForce® XLi Class IV laser therapy is applied over the affected nerve territories, where near-infrared photonic energy increases ATP production in damaged nerve cells, reduces local neuroinflammation and supports remyelination. IV nerve-support infusions deliver vitamin B12, alpha-lipoic acid and NAD+ into circulation directly, bypassing the gastrointestinal absorption limits that blunt oral supplementation in many neuropathy patients. Which of these are used, and in what sequence, depends on your testing. ### Glucose Control Is Still Part of the Work No in-clinic protocol substitutes for glycemic management. If glucose stays uncontrolled, the mechanism that damaged the nerve keeps operating, and progress made in the clinic is spent holding ground rather than gaining it. We work alongside the physician managing your diabetes rather than around them, and we do not ask patients to stop prescribed medication. Metabolic support here is additive to that care, not a replacement for it. **Can diabetic neuropathy be reversed?** That depends entirely on the stage. Nerve fibers that are damaged but still conducting can often improve in function, and symptoms frequently respond in appropriate candidates. Fibers that have already degenerated do not come back, and we will say so plainly if that is what your nerve conduction study shows. **Do I need to stop my diabetes medication to do this?** No. The protocol is designed to work alongside the care you already receive, and we do not ask patients to discontinue medication prescribed by another physician. Any change to your diabetes regimen is a conversation for the physician managing it. **How long does the protocol take?** Most patients complete an initial protocol over 8 to 12 weeks, with maintenance visits typically quarterly afterward. The exact schedule depends on which modalities your root-cause findings call for. **Does insurance cover diabetic neuropathy treatment here?** Do not assume these components are covered. Coverage is checked against your specific plan at consultation, and you are quoted before anything is scheduled. --- ## Peripheral Neuropathy - URL: https://rebuildneuropathytreatmentcenter.com/conditions/peripheral-neuropathy - Also called: Idiopathic peripheral neuropathy - Definition: Peripheral neuropathy is damage to the nerves carrying signals between the spinal cord and the limbs. When testing does not identify diabetes, compression, chemotherapy or another clear driver, the case is often labeled idiopathic. A correctable nutritional or hormonal deficiency turns up in a meaningful share of those cases once someone runs the labs. - Common signs: Symmetric numbness that began in both feet at about the same time; Pins and needles that has crept up toward the ankles over time; Burning that intensifies at rest and interferes with sleep; Reduced vibration sense at the great toe on examination; Bedsheets or socks that feel painful against the skin; Cramping or a feeling of weakness in the small muscles of the foot - Causes: Vitamin B12, B6 or folate deficiency; Thyroid hormone deficiency; Chronic alcohol use; No identified driver after a complete workup, which is what idiopathic means - How it is identified: The evaluation begins with distribution and history, since a symmetric stocking pattern that began distally points somewhere different than a single-limb complaint. Laboratory testing typically includes B12 with methylmalonic acid, folate, thyroid function, A1c and fasting glucose, and a metabolic panel, because these identify the drivers that are still correctable. Monofilament and vibration testing quantify sensory loss at the feet. Nerve conduction studies distinguish reduced amplitudes, which indicate surviving but damaged fibers, from absent potentials, which indicate fibers that are gone. - Modalities typically indicated: iv-nerve-support-infusions, class-iv-laser-therapy, hormonal-correction - Prognosis: Nutritional and hormonal neuropathy caught while the deficiency is still correctable tends to be the most responsive category we treat. Long-standing idiopathic neuropathy with significant axonal loss has a lower ceiling, and improvement there is often measured in symptom burden and stability rather than full return of sensation. Complete loss of sensory function confirmed by absent potentials on nerve conduction testing is irreversible structural damage that no conservative protocol addresses. Expectations are set against your own test results at consultation. ### Idiopathic Does Not Always Mean Unexplained Idiopathic means no cause was found, which is not the same as no cause existing. B12 deficiency, thyroid dysfunction and impaired glucose tolerance short of diabetes all produce a distal symmetric neuropathy, and all three are missed when the workup stops at a normal fasting glucose. We treat the label as a starting point for testing rather than a conclusion. If a correctable driver is present, correcting it is the treatment. ### The Workup Most Patients Have Not Had A serum B12 in the low-normal range can still be functionally deficient, which is why methylmalonic acid is checked alongside it. Thyroid function, folate, A1c and a metabolic panel round out the picture, and a nerve conduction study establishes whether the fibers are slowed or lost. This testing is ordered before a protocol is recommended, and what it returns changes which treatment is appropriate. Ordering modalities before this is done is how patients end up paying for care aimed at the wrong mechanism. ### What the Protocol Uses for Nutritional and Idiopathic Cases Where a deficiency is identified, IV nerve-support infusions deliver vitamin B12, alpha-lipoic acid and NAD+ into circulation directly, bypassing the gastrointestinal absorption limits that reduce the efficacy of oral supplementation in many neuropathy patients. Where thyroid or sex hormone deficiency is contributing, hormonal correction addresses it, because correcting the deficiency treats the driver rather than the symptom it produces. Contraindications and monitoring requirements are reviewed individually before anything is prescribed. Class IV laser therapy is applied over the affected nerve territories to increase ATP production in damaged nerve cells, reduce local neuroinflammation and support remyelination. The combination is matched to your findings, not sold as a package. ### When the Cause Stays Unknown Some cases remain genuinely idiopathic after a full workup. That does not make the nerve damage untreatable, but it does change what we can promise, because we are then supporting nerve tissue rather than removing a driver. In that situation the protocol targets the symptomatic and functional spectrum, and progress is reassessed against your baseline testing instead of assumed. Your clinician will be direct with you about which situation you are in. **My neurologist said my neuropathy is idiopathic. Is there anything left to do?** Possibly. The first step is confirming that the correctable causes were actually excluded, particularly functional B12 deficiency, thyroid dysfunction and impaired glucose tolerance. If one of those is present, it becomes the target. If none are, treatment shifts to supporting the nerve tissue itself and we will be clear about what that can and cannot do. **Why infusions instead of oral vitamins?** Oral supplementation depends on gastrointestinal absorption, which is limited in many neuropathy patients and can be further reduced by medication and age. Infusions deliver B12, alpha-lipoic acid and NAD+ into circulation directly, which removes that variable. **How soon would I know whether it is working?** The initial protocol typically runs 8 to 12 weeks, and progress is measured against the sensory testing recorded at your first visit rather than impression alone. Some patients notice changes in night symptoms earlier than that. Others do not respond, and continuing a protocol that is not producing measurable change is not something we recommend. **Do you treat neuropathy in the hands as well as the feet?** Yes, when it is part of a peripheral neuropathy picture. Hand involvement that appears without foot involvement usually suggests compression at the wrist or neck instead, which is evaluated and treated differently. --- ## Compressive Neuropathy - URL: https://rebuildneuropathytreatmentcenter.com/conditions/compressive-neuropathy - Also called: Nerve compression and entrapment neuropathy - Definition: Compressive neuropathy is nerve dysfunction caused by mechanical pressure on a nerve root or a peripheral nerve rather than by a systemic metabolic process. The pressure may come from a herniated disc, a narrowed spinal canal, or fibrotic tissue that has formed around a nerve after chronic inflammation or injury. Because a specific nerve is affected, the symptoms follow that nerve's territory instead of appearing symmetrically in both feet. - Common signs: Symptoms in one limb, or clearly worse on one side; Pain that radiates from the low back or neck down into the limb; Numbness that changes with position, bending, or how far you walk; Weakness in a specific muscle group rather than general unsteadiness; Night symptoms in the hand that ease with shaking or repositioning; A band of altered sensation that maps to one nerve distribution - Causes: Lumbar or cervical disc herniation pressing on an exiting nerve root; Spinal stenosis narrowing the space available to the nerve; Fibrotic tissue entrapping a peripheral nerve after chronic inflammation or injury; Local swelling within a confined anatomical tunnel, such as at the wrist or elbow - How it is identified: The examination maps symptoms to a specific dermatome and myotome, since compression follows anatomy in a way metabolic neuropathy does not. Positional and provocative testing helps localize the level, and walking tolerance is documented when stenosis is suspected. MRI identifies disc herniation, foraminal narrowing and canal stenosis when imaging is indicated. Electrodiagnostic testing distinguishes focal slowing or conduction block at a single site from the diffuse, length-dependent pattern of a systemic neuropathy, which matters when both are present. - Modalities typically indicated: mechanical-decompression, shockwave-therapy, class-iv-laser-therapy - Prognosis: Compressive neuropathy with a genuinely reversible mechanical component is among the more responsive presentations, particularly when the compression has not been present for years. The longer a nerve has been compressed, the more likely that axonal loss has occurred alongside the conduction block, and that portion does not recover with decompression. Cases with dense weakness, muscle wasting, or absent potentials on nerve conduction testing have a ceiling set by structural damage. Where surgery is the more appropriate answer, we will say so. ### Compression Produces a Different Pattern Metabolic neuropathy is symmetric and starts at the toes because it affects the longest fibers first. Compression affects whichever nerve is under pressure, so the pattern is focal, often one-sided, and it frequently follows a recognizable dermatome. Position matters too: symptoms that ease when you lean forward on a shopping cart, or that appear after a set walking distance, point toward stenosis rather than glucose. Getting this distinction right at the first visit determines everything that follows. ### Where the Compression Usually Sits In the lower limb, the common sources are a lumbar disc herniation or foraminal narrowing compressing a nerve root on its way out of the spine. In the upper limb, entrapment within a confined tunnel at the wrist or elbow is more typical, and cervical nerve root compression can produce overlapping symptoms. Complex cases carry more than one component at once, with a compressed root sitting on top of a metabolic neuropathy. Both have to be identified or treatment aims at half the problem. ### Decompression, Shockwave and Laser in Combination Mechanical decompression using the Antalgic-Trac® targets the compressive component directly, which is the part no infusion or supplement can reach. Chattanooga Intelect RPW 2 shockwave therapy stimulates blood flow restoration to the vasa nervorum, the nerve's own vascular supply, and breaks down fibrotic tissue that can entrap peripheral nerves after chronic inflammation. Class IV laser therapy is applied over the affected nerve territory to reduce local neuroinflammation and support remyelination in fibers that have been compressed. These are sequenced according to where your imaging and examination locate the problem. ### Red Flags That Need Emergency Care, Not a Consultation Some presentations are not a booking question at all. Sudden weakness in both legs, numbness across the groin, buttocks or inner thighs, or new loss of bladder or bowel control can mean the nerve roots at the base of the spinal cord are being crushed. That combination is a surgical emergency. Do not call this clinic and do not wait for an appointment: go to a hospital emergency department immediately, or call 911. Progressive motor weakness or a foot that has started to drop is also a surgical question rather than a conservative one, and it needs same-day medical assessment rather than a treatment protocol. Severe stenosis with bone contacting the nerve may also have a mechanical limit that decompression cannot overcome. We would rather tell you that at consultation than sell you a protocol aimed at a problem that needs an operation. **How do I know whether my neuropathy is compressive or metabolic?** Distribution is the first clue. Compression tends to affect one limb in a pattern that follows a single nerve, and often changes with position or activity. Metabolic neuropathy is symmetric and starts in both feet. Examination and, where indicated, imaging and electrodiagnostic testing confirm which you have, and some patients have both. **Is decompression the same as chiropractic adjustment?** No. Mechanical decompression using the Antalgic-Trac® applies controlled traction intended to reduce compressive load on the nerve, rather than performing a manipulation. It is used here because a compressive root cause requires a mechanical intervention. **I already had back surgery. Can I still be treated?** That depends on what was done and what your current imaging shows, so it is a consultation question rather than one we can answer generally. Post-surgical anatomy changes which mechanical approaches are appropriate, and some are ruled out entirely. **How long does treatment take once compression is confirmed?** The initial protocol typically runs 8 to 12 weeks, with maintenance visits usually quarterly afterward. Compressive cases are reassessed during that window, because a mechanical problem that is not responding to mechanical treatment needs a different plan rather than more sessions. --- ## Metabolic Neuropathy - URL: https://rebuildneuropathytreatmentcenter.com/conditions/metabolic-neuropathy - Also called: Metabolic and hormonal polyneuropathy - Definition: Metabolic neuropathy is nerve damage driven by a systemic metabolic or endocrine disturbance rather than by direct mechanical injury. Thyroid deficiency, low testosterone, estrogen deficiency, insulin resistance and prediabetes all alter the chemical environment peripheral nerves depend on. The resulting symptoms look much like other distal neuropathies, which is why the driver is only found when it is specifically tested for. - Common signs: Numbness and tingling in both feet with no diabetes diagnosis; Burning sensations alongside fatigue, cold intolerance or weight change; Symptoms that developed gradually over years rather than after an injury; Reduced sensation on monofilament or vibration testing at the feet; Muscle cramping and a general loss of strength in the legs - Causes: Hypothyroidism or inadequately treated thyroid deficiency; Testosterone or estrogen deficiency; Insulin resistance and prediabetes short of a diabetes diagnosis; Chronic inflammatory metabolic state associated with obesity and metabolic syndrome - How it is identified: Metabolic neuropathy is identified by pairing the clinical pattern with endocrine and metabolic laboratory testing. That typically means A1c and fasting glucose to catch impaired glucose tolerance, thyroid function including free T4, and morning testosterone or estradiol where the history and symptoms point that direction. B12 with methylmalonic acid is included because deficiency frequently coexists and changes the treatment plan. Monofilament and vibration testing document the sensory baseline, and nerve conduction studies are added when the extent of fiber loss needs to be quantified. - Modalities typically indicated: hormonal-correction, metabolic-optimization, iv-nerve-support-infusions - Prognosis: Where a hormonal or metabolic deficiency is identified and correctable, this is one of the more responsive root causes, and improvement sometimes follows normalization alone. The ceiling is set by how long the driver ran unaddressed and how much axonal loss accumulated in that time. Advanced degeneration does not reverse, and complete loss of sensory or motor function confirmed by absent potentials on nerve conduction testing is permanent structural damage. Correction protects what remains, which is a different goal than restoring what is gone. ### Nerve Damage Can Begin Before Diabetes Does Impaired glucose tolerance and insulin resistance affect small sensory fibers before fasting glucose crosses the diabetic threshold. Patients in that window are frequently told their labs are fine while burning feet keep them awake. An A1c and a fasting insulin tell a more useful story than a single fasting glucose does. Identifying the metabolic driver at this stage is what makes the case more treatable than it will be in five years. ### Thyroid and Sex Hormones Belong in a Neuropathy Workup Hypothyroidism produces peripheral neuropathic symptoms through mechanisms unrelated to glucose, and it is common enough that it should be excluded in every case. Testosterone and estrogen deficiency contribute in a similar way. Where a deficiency is found and corrected, the driver itself is being addressed rather than the symptom it produces, which matters when the alternative is years of symptomatic medication. Contraindications and monitoring requirements are reviewed individually before anything is prescribed. This is why hormonal correction is a modality here and not an afterthought. ### Correcting the Environment the Nerve Lives In Hormonal correction addresses thyroid, testosterone or estrogen deficiency where testing identifies it as a contributing root cause. Metabolic optimization uses physician-prescribed semaglutide together with dietary guidance to reduce the inflammatory metabolic environment that drives progressive nerve damage. Peripheral neuropathy is not an FDA-approved indication for semaglutide; it is prescribed here for the metabolic driver rather than for the nerve itself. IV nerve-support infusions supply vitamin B12, alpha-lipoic acid and NAD+ directly into circulation, which matters because metabolic patients often have absorption limits that reduce what oral supplementation delivers. All of this is physician-supervised and matched to your results. ### What Correction Does Not Fix Normalizing a hormone or improving insulin sensitivity addresses the driver, which can slow further damage and gives surviving fibers a better environment. It does not regrow axons that have already degenerated. Patients who have carried an untreated metabolic driver for many years often have a mixed picture, with some function that responds and some that does not. Your baseline testing is what tells us where that line sits in your case. **My glucose is normal. Can my neuropathy still be metabolic?** Yes. Small fiber damage can begin during insulin resistance and impaired glucose tolerance, before fasting glucose becomes abnormal. Thyroid and sex hormone deficiency also cause neuropathic symptoms with entirely normal glucose. That is why the workup here goes past a single fasting number. **Is hormone therapy required to be treated here?** No. Hormonal correction is used only where testing identifies a deficiency contributing to your neuropathy. If your thyroid and hormone panels are normal, the protocol is built from the modalities that fit what testing did find. **How is this different from a flat-fee neuropathy program?** A flat-fee program applies the same sequence of treatments to every patient regardless of cause. The Rebuild Neuropathy Protocol™ establishes the root cause first and then selects the modality combination that targets it, which means two patients with identical symptoms may receive different treatment. **Will my insurance cover the testing and treatment?** Do not assume the testing or the treatment components are covered. Coverage is checked against your specific plan at consultation, and you are quoted before anything is scheduled. --- ## Chemotherapy-Induced Neuropathy - URL: https://rebuildneuropathytreatmentcenter.com/conditions/chemotherapy-induced-neuropathy - Also called: Chemotherapy-induced peripheral neuropathy (CIPN) - Definition: Chemotherapy-induced peripheral neuropathy is nerve damage caused by neurotoxic cancer treatment rather than by a metabolic or mechanical process. Platinum agents damage the sensory neurons of the dorsal root ganglion, while taxanes and vinca alkaloids disrupt the microtubule transport that keeps long axons alive. The result is usually a symmetric glove-and-stocking pattern in the hands and feet that tracks with cumulative dose. - Common signs: Numbness and tingling that appeared in the hands and feet during treatment; Cold-triggered pain in the fingers, common after oxaliplatin; Difficulty buttoning clothing, writing, or feeling small objects; Burning in the soles that is worse at night; Loss of balance from reduced position sense in the feet - Causes: Platinum compounds such as oxaliplatin and cisplatin; Taxanes such as paclitaxel and docetaxel; Vinca alkaloids and proteasome inhibitors such as bortezomib; Cumulative dose across treatment cycles - How it is identified: The diagnosis rests on treatment history first: which agents were given, at what cumulative dose, and how the symptoms tracked against the cycles. Examination documents the glove-and-stocking distribution along with monofilament, vibration and position sense at the feet. Nerve conduction studies characterize the damage as a sensory axonal process and, more importantly, distinguish reduced amplitudes from absent potentials. We also run the deficiency workup, because B12 deficiency, thyroid dysfunction and glucose intolerance frequently coexist with CIPN and are the components that respond best. - Modalities typically indicated: class-iv-laser-therapy, iv-nerve-support-infusions - Prognosis: This is the root cause where we are most cautious about expectations. Some CIPN improves gradually over the year or so following the last dose without any intervention, and supportive treatment during that period targets symptom burden and function. Where neurotoxic agents caused permanent structural damage, particularly after long-term or high cumulative platinum exposure, the ceiling of functional recovery is limited and no regenerative protocol overcomes it. End-stage neuropathy with absent potentials on nerve conduction studies is irreversible, and we will tell you that at consultation rather than after you have paid for a protocol. ### Why Chemotherapy Damages Peripheral Nerves Neurotoxic agents reach the peripheral nervous system through different routes depending on the drug class. Platinum compounds accumulate in the dorsal root ganglion, where the sensory neuron cell bodies sit outside the protection of the blood-brain barrier. Taxanes and vinca alkaloids interfere with the microtubule transport that long axons rely on to move materials from the cell body to the nerve ending. Both mechanisms hit the longest fibers hardest, which is why the hands and feet go first. ### Timing and Coordination With Your Oncology Team If you are still in active treatment, your oncologist leads and we coordinate around that. Antioxidant supplementation during chemotherapy remains a debated question, so infusion therapy in that window is not something we begin without your oncology team's input. After treatment is complete, that constraint changes and more of the protocol becomes available. Bring your treatment history, including agents and cumulative dose, to the consultation. ### What the Protocol Targets in CIPN Class IV laser therapy is applied over the affected nerve territories, delivering near-infrared photonic energy that increases ATP production in damaged nerve cells, reduces local neuroinflammation and supports remyelination. IV nerve-support infusions deliver vitamin B12, alpha-lipoic acid and NAD+ into circulation, bypassing the gastrointestinal absorption limits that are frequently pronounced after chemotherapy. We also test for the coexisting drivers that make CIPN worse, since B12 deficiency, thyroid dysfunction and undiagnosed glucose intolerance are common in this population and are individually correctable. Those are the parts we can act on. ### Where the Ceiling Sits A portion of CIPN improves on its own in the months after the final dose, and platinum-related symptoms can also worsen for a period after treatment ends before stabilizing. Sensory neurons destroyed in the dorsal root ganglion do not regenerate, and no conservative protocol changes that. We do not offer this as a treatment that reverses chemotherapy-induced nerve damage, because it does not. What it targets is the functional spectrum that remains, and your nerve conduction testing is what tells us how much of that there is. **Can this reverse my chemotherapy-induced neuropathy?** No, and we will not tell you otherwise. Sensory neurons destroyed by neurotoxic agents do not regenerate. The protocol targets the functional spectrum that remains and any coexisting correctable driver, such as a B12 deficiency, that is compounding your symptoms. **Should I start during chemotherapy or wait until it is finished?** That decision belongs with your oncologist. Antioxidant supplementation during active chemotherapy is a debated question, so we coordinate rather than proceed independently. Supportive measures that do not raise that concern can often be discussed sooner. **It has been two years since my last infusion. Is it too late?** Not necessarily, though the natural improvement that occurs in the first year is behind you at that point. Nerve conduction testing establishes what function remains, and that result determines whether a protocol is reasonable to offer or whether we should say no. **What if my nerve conduction study shows absent responses?** Absent potentials indicate complete loss of function in those fibers, which is irreversible structural damage rather than reduced conduction. Conservative regenerative protocols do not restore complete denervation. In that situation we would not recommend starting a protocol aimed at restoring sensation. --- # MODALITIES — FULL DETAIL ## Class IV Laser Therapy - URL: https://rebuildneuropathytreatmentcenter.com/services/class-iv-laser-therapy - Device: LightForce® XLi - Category: Nerve-Directed Therapy - Session length: Set at your planning visit - Applied to: Peripheral nerves - Definition: Class IV laser therapy delivers near-infrared photonic energy at power levels high enough to reach peripheral nerve tissue below the skin surface. In neuropathy care it is applied directly over the affected nerve territories, with the intent of supporting the ATP production damaged nerve cells rely on for repair. It is aimed at the local neuroinflammation around the treated nerve rather than at masking the symptom. - How it works: Nerve conduction findings and symptom mapping identify which nerve territories to treat -> The Class IV laser head is applied directly over those territories -> Near-infrared energy is delivered with the intent of supporting ATP production inside the damaged nerve cells -> Local neuroinflammation and your symptom pattern are reassessed across a series of sessions - Suited to: Diabetic peripheral neuropathy identified before advanced axonal loss; Burning, tingling or numbness that maps to a specific nerve territory; Nutritional or hormonal neuropathy where the underlying deficiency is being corrected at the same time; Patients who want a non-injectable component alongside root-cause correction - Not suited to: End-stage neuropathy with complete loss of sensory or motor function, where nerve conduction studies show absent potentials rather than reduced amplitudes; Patients expecting laser alone to substitute for correcting a metabolic, hormonal or compressive cause; Symptoms that have not yet had a root-cause workup, since the treatment target has not been established ### How near-infrared light reaches an injured nerve Class IV lasers operate at power levels that let near-infrared wavelengths pass through skin and reach the peripheral nerve tissue underneath. The intent is to support ATP production inside damaged nerve cells, because cells with more available energy have more capacity for repair than energy-starved cells do. The same exposure is directed at the local neuroinflammation around the treated nerve. Whether the affected fibers remyelinate depends on how much viable nerve tissue is left, which is what your nerve conduction findings establish before treatment starts. ### What a session actually involves You sit or lie down and the treatment head is moved slowly over the nerve territory being targeted, usually the feet and lower legs or the hands and forearms. Most patients feel a spreading warmth and nothing sharper than that. Nothing is injected, no anesthetic is used, and you drive yourself home. Sessions are short and are scheduled as a series rather than a single visit. ### Why laser is rarely used alone Photobiomodulation gives a damaged nerve more energy to work with. It does not correct the reason the nerve was damaged in the first place. If your root cause is uncontrolled blood sugar, an untreated thyroid deficiency or a compressed lumbar nerve root, laser by itself is working downstream of the problem. In the Rebuild Neuropathy Protocol™ it is paired with the correction that addresses the cause, which is why the diagnostic workup comes first. ### Where the ceiling is Laser has the most to work with on nerves that are damaged but still viable. Diabetic peripheral neuropathy caught before advanced axonal loss, and nutritional or compressive neuropathy where the driver is correctable, are the situations that respond best. Where decades of uncontrolled diabetes or long-term chemotherapy have already produced advanced axonal degeneration, permanent structural damage limits the ceiling of functional recovery. Your clinician will tell you which category your nerve conduction findings put you in. **Does Class IV laser therapy hurt?** No. Most patients feel a broad warmth over the treated area while the head is moving. There is no needle, no incision and no recovery time, and you can drive yourself home afterward. **How many laser sessions will I need?** That depends on your root cause and how long the nerve has been symptomatic. Most patients complete an initial protocol over 8 to 12 weeks, with maintenance typically quarterly afterward. Laser sessions are scheduled inside that window alongside whichever other modalities your workup calls for. **Is laser therapy covered by insurance?** Do not assume laser therapy is covered. Coverage is checked against your specific plan at consultation, and you are quoted before anything is scheduled. **Can laser therapy reverse my neuropathy?** It is directed at the biology of an injured but living nerve: energy production, local inflammation and the condition of the myelin. In appropriate candidates that often shows up as better sensation and less burning. Where nerve conduction studies show absent potentials rather than reduced amplitudes, the damage is structural, and no conservative regenerative protocol restores it. --- ## Shockwave Therapy - URL: https://rebuildneuropathytreatmentcenter.com/services/shockwave-therapy - Device: Chattanooga Intelect RPW 2 - Category: Nerve-Directed Therapy - Session length: Set at your planning visit - Applied to: Peripheral nerves and surrounding soft tissue - Definition: Radial pressure wave therapy sends acoustic pulses into tissue through a handpiece held against the skin. In neuropathy care it is applied with the intent of improving blood flow to the nerve's own vascular supply, the vasa nervorum, and of acting on fibrotic tissue that can entrap peripheral nerves following chronic inflammation. The device used here is the Chattanooga Intelect RPW 2. - How it works: Workup determines whether poor perfusion or fibrotic entrapment is contributing to your symptoms -> Gel is applied and the RPW 2 handpiece is placed over the treatment area -> Radial pressure waves are delivered with the intent of improving blood flow to the vasa nervorum -> Repeated sessions are directed at fibrotic tissue that entraps the nerve - Suited to: Peripheral neuropathy where compromised circulation to the nerve is a documented contributor; Symptoms following a long history of chronic inflammation in the affected region; Compressive neuropathy with a soft-tissue fibrotic component; Patients already in the protocol whose nerve tissue needs better perfusion to respond - Not suited to: Nerves that are already denervated, with absent potentials on nerve conduction testing rather than reduced amplitudes; Patients with a bleeding disorder or on anticoagulation, unless cleared by your clinician at consultation; Treatment areas with active infection or a known malignancy ### The blood supply problem inside a nerve A peripheral nerve depends on the vasa nervorum, the small vessels that run alongside it and feed it. When that supply is compromised, a nerve with poor perfusion has less capacity to repair itself regardless of how well the rest of the plan is designed. Shockwave therapy is aimed squarely at that problem: improving blood flow to the nerve's vascular supply. ### Fibrosis and nerve entrapment Chronic inflammation leaves fibrotic tissue behind. Around a peripheral nerve, that fibrosis can act as a physical restriction, entrapping the nerve in tissue that no longer moves the way healthy tissue does. Radial pressure waves are directed at that fibrotic tissue. Where entrapment is part of the picture, easing it changes what the other modalities are able to accomplish. ### In the treatment room Gel is applied and the handpiece is pressed against the treatment area while the device delivers rapid pulses. Patients usually describe a firm tapping sensation, and intensity is adjusted to what you tolerate comfortably. A single area takes only minutes. There is no sedation and no downtime afterward. ### When shockwave is the right lever Shockwave earns its place when compromised perfusion or fibrotic entrapment is a real part of your presentation, which the workup establishes before anything is scheduled. It is not a substitute for correcting blood sugar, a hormone deficiency or a compressed nerve root. And where nerve conduction studies show absent potentials rather than reduced amplitudes, restoring blood flow to a nerve that no longer conducts will not bring function back. **What does shockwave therapy feel like?** Most patients describe a firm, rapid tapping against the skin. It is noticeable but not sharp, and the intensity is dialed to your tolerance rather than to a fixed setting. Nothing is injected and you leave the clinic without restrictions. **Is this the same shockwave used for kidney stones?** No. The Intelect RPW 2 is a radial pressure wave device applied through the skin at therapeutic intensity for soft tissue. In this protocol it is used to improve blood flow to the vasa nervorum and to act on fibrotic tissue, not to fragment anything. **How many shockwave sessions are typical?** Shockwave is delivered as a series rather than a single visit. It is scheduled inside the initial protocol, which most patients complete over 8 to 12 weeks, with maintenance typically quarterly afterward. The exact number depends on how many areas are being treated and how your symptoms respond. **Will shockwave alone resolve my neuropathy?** It addresses two specific problems: perfusion to the nerve's vascular supply and fibrotic entrapment. If your root cause is uncontrolled diabetes, a hormone deficiency or a compressed nerve root, those still need to be corrected. That is why the workup comes before the treatment plan. --- ## IV Nerve-Support Infusions - URL: https://rebuildneuropathytreatmentcenter.com/services/iv-nerve-support-infusions - Category: Nutrient Support - Session length: Set at your planning visit - Applied to: Systemic, delivered intravenously - Definition: IV nerve-support infusions deliver vitamin B12, alpha-lipoic acid (ALA) and NAD+ directly into circulation through a vein in the arm. Intravenous delivery bypasses the gastrointestinal absorption limits that reduce the efficacy of oral supplementation in many neuropathy patients. Infusions are physician-supervised and given in the clinic. - How it works: Labs and history establish which deficiencies and absorption problems are relevant to your symptoms -> An IV line is placed and the nerve-support formulation is started under physician supervision -> B12, alpha-lipoic acid and NAD+ enter circulation directly, bypassing gastrointestinal absorption limits -> Infusions are scheduled as a series across the initial 8 to 12 week protocol - Suited to: Documented B12 deficiency that oral supplementation has not corrected; Patients on long-term metformin or with a history of gastric surgery; Diabetic or metabolic neuropathy where nutritional support is part of the treatment plan; Idiopathic peripheral neuropathy with a nutritional component identified at workup - Not suited to: Complete denervation confirmed by absent potentials on nerve conduction studies rather than reduced amplitudes, which no infusion reverses; Patients whose labs show no deficiency and no absorption problem to correct; Anyone looking for a single infusion instead of addressing the underlying root cause ### Why the oral version often underdelivers Absorption is the limiting step. Age-related changes, gastric surgery and long-term metformin use all narrow the window for absorbing B12 in particular, and a larger oral dose does not fix a ceiling set by the gut. This is a common and often unexamined reason a patient can take supplements for years without a change in symptoms. Intravenous delivery routes around that limit entirely. ### What goes into the infusion The formulation is built around vitamin B12, alpha-lipoic acid and NAD+. These are chosen for their relevance to peripheral nerve support rather than assembled as a general wellness drip. What your specific infusion contains is decided by your clinician against your labs and your root-cause findings. Everything is prepared and run under physician supervision in the clinic. ### Chair time and what the visit looks like An IV line is placed in the arm and you sit for the length of the infusion, which varies with the formulation. You can read, work or take calls. NAD+ is infused slowly by design, because a faster rate is more likely to produce flushing, chest tightness or nausea during the infusion. Plan for an unhurried appointment rather than a quick stop. ### Fit with the rest of the protocol Infusions support nerve tissue nutritionally. They do not lower an A1c, correct a thyroid deficiency or decompress a nerve root. Inside the Rebuild Neuropathy Protocol™ they run alongside whichever corrections your workup identified, and they are often scheduled in the same visit block as laser therapy. Rebuild Therapy Infusions Center, a sibling clinic at the same street address, handles general infusion therapy; the formulations here are matched specifically to neuropathy. **Do I have to be deficient in B12 for this to help?** Labs come first. If your levels and absorption are normal, your clinician will say so and the protocol will lean on the components that fit your root cause instead. Infusions are prescribed against findings, not offered by default. **How is this different from a wellness drip?** The compounds are selected for peripheral nerve support: vitamin B12, alpha-lipoic acid and NAD+. It is prescribed as part of a neuropathy protocol built on your workup, not chosen from a menu. Every infusion is physician-supervised and given in the clinic. **Should I stop my oral supplements?** Bring the full list, including doses, to your consultation. Some oral supplements complement the infusions and some duplicate them, and that call belongs to your clinician rather than to a web page. Nothing is changed without reviewing what you are already taking. **Will insurance cover the infusions?** Coverage varies by plan, and IV infusions are one of the components where policies differ most. Some patients have partial coverage and others have none. Pricing is reviewed transparently at consultation before you commit to anything. --- ## Hormonal Correction - URL: https://rebuildneuropathytreatmentcenter.com/services/hormonal-correction - Category: Root-Cause Correction - Course length: Varies; lab-driven and reviewed at follow-up - Applied to: Systemic, endocrine - Definition: Hormonal correction treats thyroid, testosterone or estrogen deficiency where hormonal dysfunction is identified as a contributing root cause of neuropathic symptoms. It is a lab-driven intervention: the deficiency is documented, corrected under physician supervision, then re-measured. It belongs in a neuropathy protocol because an untreated thyroid or sex-hormone deficiency can be the driver of the symptoms rather than a side issue. Contraindications and monitoring requirements are reviewed individually before anything is prescribed. - How it works: Bloodwork evaluates thyroid, testosterone and estrogen status against your symptom pattern -> A deficiency identified as a contributing root cause is corrected under physician supervision -> Levels are re-measured to confirm the correction held -> The remaining modalities in your protocol are adjusted based on how symptoms respond - Suited to: Documented hypothyroidism with peripheral neuropathic symptoms; Hypogonadal patients with unexplained burning, numbness or tingling; Idiopathic neuropathy that has never had an endocrine workup; Perimenopausal and postmenopausal patients with new neuropathic symptoms - Not suited to: End-stage neuropathy with absent sensory and motor potentials on nerve conduction studies rather than reduced amplitudes; Patients whose thyroid, testosterone and estrogen levels are already normal; Anyone with a contraindication to hormone therapy identified at consultation ### The endocrine cause that gets missed An untreated hormone deficiency can be the whole explanation for a neuropathy, not a footnote to one. In hypothyroid and hypogonadal patients, neuropathic symptoms can improve with hormone normalization alone, which is a strong argument for checking before assuming. Thyroid, testosterone and estrogen are all evaluated because any of the three can be the driver. Finding one changes the entire treatment plan. ### Testing before treating Nothing is prescribed on symptoms alone. Bloodwork establishes where your levels actually sit, correction is dosed against those numbers, and levels are re-measured to confirm the correction held. If your labs come back normal, hormonal correction is not part of your protocol and your clinician will tell you so. A root-cause workup rules things out as often as it rules them in. ### What changes once the deficiency is corrected When hormone normalization was the missing piece, the rest of the protocol is working with a body that is no longer fighting an endocrine deficit. Some patients then need less from the other modalities than the original plan anticipated. Others need the full combination, because the hormone deficiency was one contributor among several. Both outcomes are common, which is why the plan is reassessed rather than sold as a fixed package. ### The limits of hormone correction Correcting a deficiency helps a nerve that can still respond. It does not undo advanced axonal degeneration from decades of uncontrolled diabetes or long-term chemotherapy, where permanent structural damage limits the ceiling of functional recovery. Hormone therapy also carries its own monitoring requirements and is not appropriate for every patient, which is assessed at consultation. Physician supervision continues for as long as you are on it. **How would I know if my neuropathy is hormonal?** You usually would not, because the symptoms feel the same as any other peripheral neuropathy. That is why thyroid, testosterone and estrogen are checked as part of the root-cause workup rather than only when something suggests them. The labs answer the question; the symptom pattern alone does not. **Can correcting a hormone deficiency be enough on its own?** Sometimes. Where a thyroid or sex-hormone deficiency turns out to be the driver, correcting it can be enough on its own. In other patients the deficiency is one contributor among several and the rest of the protocol is still needed, which is why symptoms and labs are both reassessed after correction. **Will I be on hormone therapy permanently?** That depends on why the deficiency exists. Some causes are correctable and some require ongoing replacement, and your clinician will be direct with you about which applies. Either way it stays physician-supervised and is reviewed at follow-up rather than set and forgotten. **Is hormone testing part of the initial workup?** Endocrine status is part of establishing the root cause, which is the first step in the Rebuild Neuropathy Protocol™ before any modality is scheduled. What is ordered is matched to your history and presentation. Findings are reviewed with you at consultation. --- ## Metabolic Optimization - URL: https://rebuildneuropathytreatmentcenter.com/services/metabolic-optimization - Category: Root-Cause Correction - Course length: Varies; runs across the protocol and beyond - Applied to: Systemic, metabolic - Definition: Metabolic optimization targets the inflammatory metabolic environment that drives progressive nerve damage in diabetic and metabolic neuropathy. It combines physician-prescribed semaglutide with dietary guidance. The purpose is to reduce the process that keeps damaging the nerve while the other modalities work on the nerve itself. - How it works: A1c, metabolic labs and diet history establish which inflammatory drivers are in play -> Semaglutide is prescribed under physician supervision where appropriate -> Dietary guidance targets the metabolic environment between visits -> Labs are repeated to confirm the drivers are moving in the right direction - Suited to: Diabetic peripheral neuropathy with an A1c that is not yet controlled; Metabolic neuropathy where weight and inflammation are documented contributors; Patients whose symptoms have been progressing rather than holding steady; Patients willing to make dietary changes alongside the prescribed component - Not suited to: Advanced axonal loss with absent potentials on nerve conduction studies rather than reduced amplitudes, where slowing further damage cannot restore fibers already lost; Patients with a contraindication to semaglutide, which is assessed at consultation; Anyone expecting the prescribed component to work without the dietary side of it ### Nerve damage that is still in progress Patients often assume their neuropathy is a fixed injury they now have to live around. In diabetic and metabolic neuropathy it is usually more accurate to say the damage is ongoing. The inflammatory metabolic environment that produced the first symptoms keeps producing more, which is why symptoms progress rather than plateau. This component exists to reduce that driver. ### The prescribed component and dietary guidance The prescribed component is semaglutide, given under physician supervision and only where your workup supports it. Dietary guidance is the other half, and it is specific rather than a printed handout, because what you eat sets the metabolic environment your nerves sit in every day between visits. Neither half is a stand-alone answer. Both are directed against what your labs actually show. ### Coordinating with the physician managing your diabetes If you are already being treated for diabetes, that care continues. This component is aimed at the inflammatory metabolic environment driving nerve damage; it does not replace your endocrinologist or primary care physician. Bring your current medication list and your most recent A1c to consultation so the plan is built on top of what you are already doing rather than in conflict with it. ### How quickly to expect change Metabolic change is slower than the symptom relief patients notice from laser or infusions, and early on it is measured in labs rather than in how your feet feel this week. Most patients complete an initial protocol over 8 to 12 weeks, with maintenance typically quarterly afterward, and the metabolic work often continues past that window. The return is a lower rate of ongoing damage, which protects whatever recovery the rest of the protocol achieves. **Is semaglutide here for weight loss or for my nerves?** The target is the inflammatory metabolic environment that drives progressive nerve damage in diabetic and metabolic neuropathy. Peripheral neuropathy is not an FDA-approved indication for semaglutide; it is prescribed here for the metabolic driver rather than for the nerve itself. Weight change is often part of how that environment improves. Whether it is appropriate for you is decided at consultation. **Do I really have to change my diet?** Yes, if metabolic drivers are part of your root cause. Diet is what sets the environment your nerves live in between appointments, and no prescription compensates for leaving it unchanged. The guidance is built around what you actually eat rather than a generic plan. **Can this stop my neuropathy from getting worse?** Reducing the inflammatory metabolic driver is how progression is addressed, and in appropriate candidates that often slows the decline that brought them in. It cannot restore fibers already lost to advanced axonal degeneration. Your nerve conduction findings tell your clinician which part of that spectrum you are on. --- ## Mechanical Decompression - URL: https://rebuildneuropathytreatmentcenter.com/services/mechanical-decompression - Device: Antalgic-Trac® - Category: Structural Decompression - Session length: Set at your planning visit - Applied to: Cervical and lumbar spine - Definition: Mechanical decompression uses the Antalgic-Trac® to apply controlled traction aimed at relieving spinal nerve root compression. It is included in neuropathy care because a compressive component is often present alongside other root causes in complex cases. Treatment is non-surgical and delivered on a table under clinician direction. - How it works: Examination, history and any available imaging establish whether a compressive component is present -> Traction parameters are set for the spinal segment involved -> The Antalgic-Trac® applies controlled decompression across a timed session -> Symptom response guides how much of your presentation was compressive - Suited to: Compressive neuropathy with a reversible mechanical component; Radiating leg or arm symptoms that follow a nerve root distribution; Complex cases where a compressive contributor sits alongside a metabolic one; Patients seeking a non-surgical option before considering surgical consultation - Not suited to: End-stage neuropathy showing absent potentials on nerve conduction studies rather than reduced amplitudes; Patients whose examination shows no compressive component to treat; Anyone with a spinal condition that makes traction inappropriate, which is screened at consultation ### When the symptom is in the foot but the problem is in the spine A compressed nerve root refers symptoms down the whole distribution of that nerve. Patients arrive certain the problem is in their feet because that is where they feel it. The distinction matters because a compressive neuropathy and a metabolic one call for completely different treatment. Sorting this out is part of the root-cause workup rather than something assumed at the first visit. ### Compressive, metabolic, or both In complex cases the answer is often both. A patient can have diabetic nerve damage and a compressed lumbar nerve root at the same time, and treating only one leaves residual symptoms that get blamed on the treatment. Mechanical decompression addresses the compressive component. The other modalities in the protocol handle what remains. ### On the table You are positioned on the Antalgic-Trac® and traction is applied gradually, with parameters set by your clinician for the segment involved. Most patients find it comfortable and some find it relieving during the session itself. There is no sedation, and you walk out under your own power. Decompression is delivered as a series rather than a single visit. ### The reversible component, and the ceiling Decompression is most useful in compressive neuropathy where there is a reversible mechanical component, which is the situation conservative care is built for. If examination and imaging show compression that is fixed or severe, your clinician will say so and refer appropriately rather than run a series that will not help. And where nerve conduction studies show absent potentials rather than reduced amplitudes, the downstream damage is structural, and relieving compression will not restore a nerve that no longer conducts. **How do I know whether my neuropathy is compressive?** The symptom pattern is the first clue: compressive symptoms tend to follow a nerve root distribution and often change with position or activity. Examination, history and any imaging you already have fill in the rest. This is established during the root-cause workup before decompression is scheduled. **Is this the same as a chiropractic adjustment?** No. An adjustment applies a manual thrust to a joint. Mechanical decompression applies controlled traction on a table across a timed session, with parameters set for the segment being treated, aimed at relieving compression on a spinal nerve root. **Do I need an MRI before starting?** Bring any recent imaging to your consultation, since it makes the picture clearer. Where imaging is needed and you do not have it, your clinician will tell you that before a decompression series begins rather than after. Nothing is scheduled on assumption. **How does decompression fit with the rest of the protocol?** It handles the compressive component while laser, shockwave, infusions, hormonal correction or metabolic optimization address whatever else the workup identified. In complex cases more than one cause is usually in play. Most patients complete an initial protocol over 8 to 12 weeks, with maintenance typically quarterly afterward. --- # SERVICE AREA ## Pompano Beach - URL: https://rebuildneuropathytreatmentcenter.com/pompano-beach-neuropathy-treatment - Pompano Beach sits directly south of the clinic, and Federal Highway connects the two without an interstate ramp in between. Many people with neuropathy have already tried gabapentin, a topical cream, or a supplement regimen that did not hold. The Rebuild Neuropathy Protocol™ starts somewhere else: identifying which root cause is actually driving the nerve damage, before any modality is selected. What follows is built around that finding rather than around a package. - Getting here: From central Pompano Beach, Federal Highway runs north across Atlantic Boulevard and Copans Road and delivers you to 3320 N Federal Hwy without a turn onto I-95. Patients coming from the western neighborhoods usually take Copans Road or Sample Road east to US-1, then head north. Call (754) 547-2350 if you want the route confirmed before you leave. ## Deerfield Beach - URL: https://rebuildneuropathytreatmentcenter.com/deerfield-beach-neuropathy-treatment - Deerfield Beach is the clinic's neighbor to the north, close enough that the drive is rarely the deciding factor. What usually prompts the call is a sentence heard somewhere else: that neuropathy is progressive, and the choice is medication or nothing. That is accurate for some presentations and wrong for others. The difference comes down to the root cause and how far the nerve damage has progressed, which is what the evaluation is for. - Getting here: Take Federal Highway south from Hillsboro Boulevard and the clinic is a short run down at 3320 N Federal Hwy. Coming off I-95, exit at Hillsboro Boulevard and head east to US-1 before turning south. From the western side of Deerfield Beach, Sample Road east to Federal Highway works just as well. ## Boca Raton - URL: https://rebuildneuropathytreatmentcenter.com/boca-raton-neuropathy-treatment - Boca Raton residents often reach a neuropathy clinic having already worked through the standard steps: a neurologist visit, a nerve conduction study in hand, and a year or longer of managing symptoms with a prescription. The question that follows is whether anything addresses the nerve tissue itself rather than the pain signal. That is the question the Rebuild Neuropathy Protocol™ was built around. - Getting here: I-95 south to the Hillsboro Boulevard exit, then east to Federal Highway and south into Lighthouse Point, is the most direct route from Boca Raton. US-1 carries the name Federal Highway on both sides of the county line and runs the whole distance if you would rather stay off the interstate. Plan on roughly twenty minutes outside of peak season traffic. ## Coconut Creek - URL: https://rebuildneuropathytreatmentcenter.com/coconut-creek-neuropathy-treatment - Coconut Creek sits inland, west of the coastal corridor, and the drive east is the variable most patients weigh before booking. It helps to know what the trip buys. Neuropathy that traces back to metabolic dysfunction behaves differently from compressive or nutritional neuropathy, and it responds to a different combination of modalities. Sorting out which one you have is the first appointment's only job. - Getting here: Sample Road and Copans Road both run east from Coconut Creek to Federal Highway, and either puts you close to 3320 N Federal Hwy. Sample Road is generally the cleaner run. If you use Florida's Turnpike or the Sawgrass Expressway, come off toward Sample Road and continue east to US-1. ## Fort Lauderdale - URL: https://rebuildneuropathytreatmentcenter.com/fort-lauderdale-neuropathy-treatment - Fort Lauderdale is one of the longer drives in this service area, and patients who make it usually have a specific reason. Often it is a compressive component, nerve root involvement sitting underneath a peripheral neuropathy, that has not been addressed anywhere else. Sometimes it is simply that they want a diagnostic workup instead of another prescription. The consultation is where you find out whether the drive is worth repeating. - Getting here: From downtown Fort Lauderdale and the beach neighborhoods, Federal Highway runs north through Oakland Park and Pompano Beach directly to the clinic. From the western side of the city, I-95 north to Copans Road or Sample Road and then east is usually faster. Allow roughly half an hour either way. ## Oakland Park - URL: https://rebuildneuropathytreatmentcenter.com/oakland-park-neuropathy-treatment - The symptoms that eventually bring people to a neuropathy clinic usually start small. Burning in the feet at night. A sock that feels bunched when it is not. Toes that have gone quiet. Oakland Park patients who come in at that stage generally have more to work with than those who wait until balance and gait are affected, and the protocol reflects that difference. - Getting here: Federal Highway runs north out of Oakland Park through Pompano Beach and into Lighthouse Point without requiring the interstate at any point. If US-1 is heavy, take I-95 north and come east on Copans Road or Sample Road instead. The address is 3320 N Federal Hwy. ## Coral Springs - URL: https://rebuildneuropathytreatmentcenter.com/coral-springs-neuropathy-treatment - Coral Springs sits at the western end of the Sample Road corridor, which happens to run almost directly to the clinic. Patients making that drive frequently share the same diagnosis: idiopathic peripheral neuropathy. Idiopathic means the cause was not found. That is a different statement from the cause not existing, and a good share of the evaluation is spent in that gap. - Getting here: Sample Road runs east from Coral Springs through Coconut Creek and Pompano Beach and meets Federal Highway near the clinic. Atlantic Boulevard east to US-1 and north is the alternative when Sample is backed up. From the far western side of the city, the Sawgrass Expressway across to Sample Road avoids a stretch of lights. ## Parkland - URL: https://rebuildneuropathytreatmentcenter.com/parkland-neuropathy-treatment - Parkland is the farthest northwest point in the clinic's service area, and for most residents the drive east on Hillsboro Boulevard is already a familiar one. The complaint that tends to prompt the call is not pain. It is balance: reaching for a wall in a dark hallway, or noticing that the ground has stopped reporting back through the feet. Proprioceptive loss is a sensory nerve problem, and it gets its own place in the evaluation. - Getting here: Hillsboro Boulevard runs east from Parkland across the top of Broward County to Federal Highway. Turn south there and the clinic is a short distance down at 3320 N Federal Hwy. Plan on roughly half an hour. Call (754) 547-2350 if you want the route confirmed against where in Parkland you are starting from. --- # FREQUENTLY ASKED QUESTIONS ## About neuropathy **What causes peripheral neuropathy?** Diabetes is the most common driver, but it is far from the only one. Nerve damage also follows B12 and other nutritional deficiencies, thyroid and sex hormone deficiency, chemotherapy, mechanical compression of a nerve root, and metabolic inflammation. A large share of cases are labeled idiopathic, which frequently means the workup stopped before a cause was identified. **Why do symptoms usually start in the feet?** Peripheral neuropathy typically affects the longest nerves first, and the longest nerves in the body run to the toes. That is why burning, pins and needles and numbness usually begin in the feet and move upward over time, reaching the hands later in a length-dependent pattern. A symptom pattern that does not follow that distribution suggests a different mechanism and changes the workup. **Is neuropathy progressive?** Untreated, it often is, particularly while the underlying driver is still active. Uncontrolled blood sugar, an uncorrected deficiency or an unresolved compression continues to damage axons over time. Progression is not inevitable. When the cause is identified and addressed, the trajectory can change, which is why timing matters more than most patients are told. **What is the risk of leaving it alone?** The most immediate risk is loss of protective sensation. When the feet stop reliably reporting pressure, heat and injury, wounds go unnoticed, and that is the pathway to foot ulcers and, in diabetic patients, more serious complications. Balance also degrades as position sense fades, which raises fall risk. A regular foot exam is a reasonable minimum even if you do not pursue treatment. ## The protocol **What is The Rebuild Neuropathy Protocol™?** It is a sequence: root-cause diagnosis first, then a combination of modalities selected to match the cause identified. Six modalities are available: Class IV laser, shockwave, IV nerve-support infusions, hormonal correction, metabolic optimization and mechanical decompression. Most patients receive some of them, not all. The initial course usually runs eight to twelve weeks. **How is this different from a fixed-session neuropathy program?** A fixed program prices a set number of sessions before the cause of the neuropathy has been established, which means the treatment is decided in advance and the diagnosis has to fit around it. Here the order is reversed. The diagnostic visit determines which modalities are indicated, and the plan and its cost follow from that. Patients who are not good candidates are told so before anything is scheduled. **Which root causes does the protocol address?** The protocol is organized around five categories: diabetic, peripheral including idiopathic and nutritional, compressive, metabolic, and chemotherapy-induced. Many patients have more than one operating at the same time, which is common and is one reason the modality combination varies so much between patients. The diagnostic visit is where those are sorted out and ranked. **How soon might I notice a change?** It varies with the cause and with how much nerve tissue remains viable. Patients whose neuropathy is driven by a correctable deficiency or a hormonal problem sometimes notice change within the first few weeks of correction. Structural and metabolic cases usually take longer, and peripheral nerve regeneration is measured in months rather than days. Progress is reassessed against your baseline testing during the course rather than judged on impression alone. ## Treatment sessions **How long is a treatment session?** It depends on which modalities your protocol includes. Laser and shockwave applications over the affected nerve territories are relatively short. IV nerve-support infusions take longer, since B12, alpha-lipoic acid and NAD+ are delivered by slow infusion in clinic. Your schedule is set at the planning visit, so the time commitment is clear before you agree to it. **How often would I come in?** The initial course typically runs over eight to twelve weeks, with session frequency set by the modality mix. Laser and shockwave are usually scheduled more frequently than infusions. After the initial course, most patients move to maintenance visits that are typically quarterly. **Are there side effects?** Each modality has its own profile, reviewed with you before treatment begins. Shockwave can leave transient soreness or mild redness over the treated area. IV infusions carry the usual risks associated with peripheral IV access, and some patients report flushing or a warm sensation during the infusion. Peptides and hormone therapy are prescribed and monitored under physician supervision, with the risks discussed individually. **Can I keep taking my current medications?** Bring the full list, including supplements and doses, to the first visit. Many patients continue gabapentin, pregabalin, duloxetine or other prescribed medication through the protocol, since those treat symptom transmission rather than the underlying cause. Any change to a prescribed medication is a decision for the physician who prescribed it. Do not stop or adjust medication on your own. ## Practical **What does treatment cost?** Cost depends entirely on which modalities your diagnosis calls for, so nothing is quoted before the evaluation. Pricing for the recommended components is reviewed transparently at consultation, itemized rather than bundled into a single flat fee. You will know what the protocol involves and what it costs before anything is scheduled. **Is any of this covered by insurance?** Do not assume these components are covered. Coverage is checked against your specific plan at consultation, and you are quoted before anything is scheduled. Bring your insurance information to that visit so it can be reviewed against your own plan rather than discussed in general terms. **Do I need a referral to be seen?** No referral is needed to book a consultation. If a neurologist, primary care physician or podiatrist has already worked you up, bring what they produced. Prior nerve conduction studies, EMG reports and recent labs shorten the process and reduce duplicated testing. We coordinate with the physicians already managing your care where that is useful. **I would be traveling in for treatment. Is that practical?** It can be, but the protocol requires repeated in-clinic sessions across roughly eight to twelve weeks, and the infusion, laser and shockwave components cannot be done remotely. Patients traveling from elsewhere in Broward or south Palm Beach County generally manage the schedule without difficulty. If you are coming from further away, raise it at the consultation so the schedule can be planned realistically before you commit.